Urinary System

Anatomy and Physiology II

BIO 232

Path of Urine

      Kidney (Anatomy)

    Retroperitoneal position - between dorsal body wall and the parietal peritoneum - superior lumbar position

    From 12 thoracic vertebra to the third lumbar vertebra

    Adult kidney weighs about 150 gram (5 ounces)

Path of Urine

      Ureters

    Muscular tubes that convey urine to the bladder by peristalsis

      Urinary Bladder - hollow muscular organ

    600ml of urine

      Urethra

    Extends from the urinary bladder to external opening

Kidney Anatomy

 

 

 

 

Internal Anatomy of the Kidney

      Renal cortex - light in color & granular in appearance

      Renal medulla - darker reddish-brown color

     Contains cone-shaped - medullary or renal pyramids

     Renal columns - inward excursions of cortical tissue - separate the pyramids

      Renal pelvis - flat, funnel-shaped tube - continuous with the ureters

     See two or three major calyces - receive the urine from collecting ducts

Internal Anatomy of the Kidney

 

 

 

 

 

 

Nephrons

      Over 1 million nephrons or kidney tubules in each kidney

      Thousands of collecting ducts, each collects urine from several nephrons

      Each nephron

     Glomerular capsule (Bowman's capsule)

    Surrounds the glomerulus (tuft of capillaries)

    Together - renal corpuscle

     Proximal convoluted tubule (PCT)

     Loop of Henle

     Distal convoluted tubule (DCT)

Structure of a Nephron

 

 

 

 

 

 

Capillary Beds of the Nephron

      Glomerulus - specialized for filtration

     Fed by an afferent arteriole and drained by an efferent arteriole

      Peritubular capillaries

     Arise from the efferent arteioles draining the glomerulus

     Adapted for absorption - they cling closely to the renal tubules

Vasa recta - long straight bundles of capillaries that extend deep into the medulla paralleling the courses of the longest loops of Henle

Function in helping to concentrate the filtrate

Vasculature of Nephrons

 

 

 

 

 

 

Juxtaglomerular Apparatus

      DCT lies against the afferent arteriole feeding the glomerulus

      In the arteriole wall are juxtaglomerular cells (JG) - act as mechanoreceptors that detect blood pressure

      Macula densa - group of tall, closely packed cells of the DCT - lie adjacent to the JG cells

     These cells are Osmoreceptors - respond to solute content of the tubule lumen

     The above two sets of cells - regulate rate of filtrate formation & systemic blood pressure

Filtration Membrane

      Between the blood and the interior of the glomerular capsule

      Three layers

1. Fenestrated endothelium of the glomerular capillaries

2. Visceral membrane of the glomerular capsule made of podocytes

3. Intervening basement membrane composed of fused basal laminas of the other layers

Filtration Membrane

 

 

 

Glomerular Filtration

      Passive, nonselective whereby fluids & solutes are forced through a membrane by hydrostatic pressure - forming filtrate

      All molecules smaller than 3nm in diameter pass from the blood into the renal tubules

    Water, glucose AAs & nitrogenous wastes

      Glomerular filtration rate (GFR)

    120-125ml/min 97.5 L/hr or 180 L/day (47 gallons/day)

Renin-Angiotensin Mechanism

      Triggered by JG cells - release renin

      Renin is an enzyme that acts of angiotensinogen (plasma) - to get angiotensin I

      In the lungs angiotensin i is converted into angiotensin II by an ACE

      Angiotensin II is a powerful vasoconstrictor

     Also triggers release of aldosterone by the adrenals

     Aldosterone reclaims sodium and water fallows - increased blood volume - increased BP

Renin Release Triggered By

    Reduced stretched  of the JG cells - low BP

    Stimulation of JG cells from the macula densa cells

   They release less vasoconstrictor chemical

    Sympathetic input

    Angiotensin II stimulation of JG cells

Tubular Reabsorption

      Total solute concentration of filtrate begins at 300 mOsm

      Isotonic to plasma

      Osmosis cannot occur unless the conc of plasma and filtrate are altered by active transport

Reabsorption in Proximal Convoluted Tubule

      Cell of the PCT have NA+/K+ pumps

      Pumps create a concentration gradient that favors diffusion of Na+ across the apical membranes into the cells of the PCT

      Na+ is extruded into the surrounding tissue fluid by the pumps

      This creates an electrical potential across the wall of the tubule

      CL- is passively drawn toward the high  Na+ concentration of the tissue fluid

Reabsorption in Proximal Convoluted Tubule

      Now an osmotic gradient is established - osmosis occurs

      65% of salt & water reabsorbed by PCT

      About 20% of salt & water reabsorbed by loop of Henle

Reabsorption of Glucose and Amino Acids

      Glucose & AAs recover by secondary active transport

       Mediated by membrane carriers that co-transport Na+

Reabsorption By PCT

 

 

 

 

 

 

 

Countercurrent Multiplier System

     Ascending limb of the loop of Henle

     Na+,  K+ &  Cl- passively diffuse from the filtrate into the cells of the ascending loop ( ratio of 1:1:2)

    Na+ - actively transported to tissue fluid by Na+/K+ pump

    CL- follows

    K+ diffuses back into the filtrate

Countercurrent Multiplier System

     Ascending limb of the loop of Henle

    Thin segment - nearest top of loop

    Thick segment - varying lengths - active transport of Na+

    Walls of ascending limb are not permeable to water

Ascending Limb Structure

      Thus the filtrate - increasingly dilute as it ascends toward the cortex

      In contrast - tissue fluid around the loop in medulla - more conc

      Fluid leaving the loop - 100 mOsm - hypotonic

Countercurrent Multiplier System

     Descending limb of the Loop of Henle

    Deeper regions of the medulla - 1200 - 1400 mOsm

    To reach these conc - active Na+ transport ascending limb

    Accumulates in the tissue fluid of the medulla

Countercurrent Multiplier System

      Descending limb- impermeable to passive diffusion of salt

      Permeable to water

    Surrounding interstitial fluid - hypertonic

    Water drawn out by osmosis

      As tubular fluid descends to the tip of the loop the concentration of the fluid is increased & the volume decreased

Countercurrent Multiplier System

     Countercurrent multiplication

    Countercurrent flow (flow in opposite direction) in the ascending and descending limbs

   The close proximity of the limbs allows interaction

Countercurrent Multiplier System

      Conc of filtrate in descending limb reflects conc of the surrounding tissue fluid

      Conc of the tissue fluid - raised - active transport - ascending limb

      This is a positive feedback system

      More salt excreted by the ascending limb - more concentrated will be the fluid delivered to it by the descending limb

      Multiplies the conc of the tissue fluid & the descending limb fluid

Countercurrent Multiplier System

 

 

 

 

 

 

 

Vasa Recta

      For the CCMS to work - extruded salt must remain in the tissue fluid while most of the water leaves

      Vasa recta accomplishes this

      Peritubular capillaries which run with the Loops of Henle

      Maintains the hypertonicity of the renal medulla

Countercurrent Exchange

      Salt & urea - high conc in medullary tissue fluid diffuses into blood as it descends into the loops of the vasa recta

      Then the solutes diffuses out of the ascending vessels & back into the descending vessels (conc is lower here)

Countercurrent Exchange

      Salt & urea - high conc in medullary tissue fluid diffuses into blood as it descends into the loops of the vasa recta

      Then the solutes diffuses out of the ascending vessels & back into the descending vessels (conc is lower here)

Countercurrent Exchange

      Thus solutes are cycled  & trapped in the medulla

      Solute conc are equal on the inside &outside since the walls of the vasa recta are freely permeable

      Colloid osmotic pressure in the vasa recta is higher than in the interstitial fluid

      Get osmotic movement of water at both ends of the vasa recta

Collecting Duct & ADH

      Filtrate entering collecting duct is hypotonic

      Surrounding medulla is hypertonic

      Some reabsorption of Na+ and secretion of K+ - cortical region

      Wall of duct - water channels

      Water channels produced as proteins within the membranes of vesicles that bud from the Golgi

      In the absence of ADH - vesicles in cytoplasm of the collecting duct cells

Collecting Duct & ADH

      When ADH binds to receptors

      Then C-AMP mediated fusion of vesicles - exocytosis

      New water channels

      Collecting duct now more permeable to water

      When ADH drops - water channels removed by endocytosis

      Thus water channels are recycled

Control of ADH Secretion

      ADH controlled by the hypothalamus

      Osmoreceptors in hypothalamus detect inc blood osmotic pressure

      ADH secreted

      During dehydration

      ADH secreted & collecting duct reabsorbs more water

Role of Aldosterone in Na+/K+ Balance

      90% of filtered Na+ & K+  reabsorbed

      Na+ & K+ reabsorption controlled by aldosterone

      No aldosterone - 80% of remainder of Na+  still absorbed - DCT

      Thus only 2% Na+ excreted (30 g /day)

      High Aldosterone - all Na+ reabsorbed

Role of Aldosterone in Na+/K+ Balance

      Potassium Secretion

      90% of filtered K+ - reabsorbed early

      No aldosterone - all of filtered K+ that remains is reabsorbed

      Aldosterone - stim secretion of K+ from peritubular capillaries to DCT & collecting duct

      Aldosterone high - 50 time more K+ excreted

Control of Aldosterone Secretion

      A rise in blood K+ levels - directly stimulates secretion of aldosterone

      Renin-Angiotensin system

Nervous Control of Urination (Micturition)

      When urinary bladder full of urine

      Stretch receptors from bladder send impulses to the spinal cord

      Spinal cord sends impulses back to bladder - reflex contraction

      Each contraction further stimulates stretch receptors

      After several contraction - refractory - stops for minutes to 1 hr

      Then cycle is repeated

Nervous Control of Urination (Micturition)

     At some point - internal sphincter is stretched - urine forced into urethra

     Now 2nd stretch reflex

    Inhibits spinal MNs that maintain tonic external sphincter contraction

    Once urination begun - positive feedback maintains flow

Voluntary Control of Urination

      11/2 - 3 yrs of age

      First a series of bladder contractions - signal brain

      Voluntary initiation - descending pathway that inhibits MN of the external sphincter & surrounding muscle of the pelvic floor

      Urine enters the urethra - positive feedback

      Reflex emptying of bladder assisted by muscles of the lower abdomen