Cell-Cell Communication

Paracrine Factors & Cell Surface Receptors

 

Paracrine vs. Juxtacrine Interactions

    Juxtacrine Interactions - cell membrane proteins on one cell surface interact with receptor proteins on adjacent cell surfaces

    When proteins synthesized by one cell can diffuse over small distances to induce changes in neighboring cells - paracrine interactions

  Diffusible proteins are called paracrine factors or growth and differentiation factors

Mechanisms of Inductive Interaction

 

 

 

 

 

Fibroblast Growth Factors

     Fibroblast growth factor (FGF) - family of over 12 structurally related members

     These activate a set of receptor tyrosine kinases called fibroblast growth factor receptors (FGFRs)

     When FGF binds - dormant tyrosine kinase is activated - it phosphorylates certain proteins within the responding cell

     The proteins are now activated & can perform new functions

   E.g. Angiogenesis, mesoderm formation & axon extension

FGF Expression

 

 

 

 

The Hedgehog Family

    Paracrine factors - used by embryo to induce particular cell types and to create boundaries between tissues

    Vertebrates have at least 3 homologues of Drosophila hedgehog gene

  sonic hedge (shh)

  desert hedge (dhh)

  indian hedgehog (ihh)

The Hedgehog Family

    dhh - expressed in Sertoli cells of the testes

    ihh - expressed in the gut and in cartilage - also important in postnatal bone growth

The Hedgehog Family

     Shh - most widely used - made by notochord -

   responsible for patterning the neural tube - such that motor neurons are formed from the ventral  neurons & sensory neurons are formed from the dorsal neurons

   Also patterns the somites such that the part closest to the notochord becomes the cartilage of the spine

   Mediates left-right axis in chicks, anterior-posterior axis in limbs, induce regional specific differentiation of the digestive tube & induce feather formation

The Wnt Family

    Cystine rich glycoproteins - 15 members

    Wnt 1 - active in inducing the dorsal cells of the somites to become muscle

    Also establishes polarity of limbs

    Also involve in urogenital system development

The TGF-b Superfamily

     30 structurally related members

   TGF - transforming growth factor

     Involved in many interactions from mammalian sex determination, regulating cell division, epithelial cell branching into ducts of kidneys, lungs and salivary glands

     BMP members - bone formation, cell division, apoptosis (programmed cell death), cell migration & differentiation,  etc.

Cell Surface Receptors

    Receptors that span the entire membrane that bind paracrine factors (the ligand)

    This binding brings about a cascade of interacting proteins that transmit a signal through a pathway from bound receptor to the nucleus

  Called signal transduction pathways

 Overview of Signal Transduction Pathways

     Ligand binds extracellular region of receptor - induces a conformational change ( shape change) of the receptor - transmitted to the cytoplasmic domain

     This change gives the cytoplasmic domain enzymatic activity

   Usually a kinase activity - use ATP to phosphorylate proteins

     Now the phosphorylated proteins become active

     Eventually a cascade of phosphorylation activates a dormant transcription factor

     This then activates or represses a particular set of genes

The RTK-Ras Pathway

     Receptor tyrosine kinase is dimerized by the ligand

   Ligands - fibroblast growth factor, epidermal growth factors, plateletl-derived growth factors & stem cell factor

   Each can only bind one or a small set of ligands

     Receptors undergo a conformational change - activating it

   Can also now dimerize with another RTK

   They now phosphorylate each other

     Now recognized by adapter protein

   Acts a bridge that links the phosphorylated RTK to an intracellular signaling system

The RTK-Ras Pathway

     Adapter protein activates GNRP (guanine nucleotide releasing factor)

     GNRP phosphorylates Ras G protein

     GAP (GTPase activating protein) protein can now hydrolyze  the phosphate bond - returns Ras to inactive state

     Active Ras activates protein kinase C (PKC) - this phosphorylates a series of kinases

     Eventually, activated ERK kinase alters gene expression by phosphorylating certain transcription factors (determines which will be transcribed)

   Also translation factors can be activated to alter the level of protein synthesis

The RTK-Ras Pathway

 

 

 

 

 

The JAK-STAT Pathway

     Casein gene activation by prolactin from the ant. pituitary

     This gene is activated during the last phase of mammary gland development

     Prolactin causes dimerization of prolactin receptors  in the mammary duct epithelial cells

     JAK 2 protein is “hitched” to the receptors - these phorphorylate each other & the dimerized protein -  this activates their kinase activity

     This kinase activity now adds a phosphate to a tyrosine residue (Y) of STAT 5 protein (for casein)

     STAT can now dimerize - now translocated into the nucleus - binds specific DNA region

     Now in combination with other transcription factors (already their) - activates transcription of the casein gene

The JAK-STAT Pathway

 

 

 

 

 

 

Wnt Pathway

     Wnt protein binds with a receptor which is a member of the Frizzled family of proteins

     The Frizzled protein then activates Disheveled, allowing it to became an inhibitor of Glycogen synthase kinase 3 (GSK-3)

     GSK-3 if it were active - prevents dissociation of b-catenin from the APC protein

     Therefore by inhibiting GSK-3, the Wnt signal frees b-catenin to associate with LEF protein to become an active transcription factor

Wnt Pathway

 

 

 

 

Hedgehog Pathway

     Hedgehog family of proteins bind a receptor called Patched

   Not a signal transducer - bound to a signal transducer called smoothened protein

     No Hedgehog protein

   Patched protein inhibits smoothened protein

   In the absence of hedgehog binding to Patched - Ci protein is tethered to microtubules

   PKA and slimb proteins can now cleave Ci into a transcriptional repressor that blocks transcription of certain genes

Hedgehog Pathway - No Hedgehog Protein

 

 

 

 

 

Hedgehog Pathway - Presence of Hedgehog Protein

     Hedgehog binds to Patched - conformation changes - releasing inhibition of Smoothened protein

     Smoothened protein now releases Ci from mirotubules (may add phosphates to Cos2 & Fused proteins

     Also inactivates the cleavage proteins PKA & Slimb

     The Ci protein now enters the nucleus - binds a CBP protein & act as a transcription activator for certain genes

Hedgehog Pathway - Presence of Hedgehog Protein